Advair Diskus: Inhaling for Better Breathing
How it works - Mechanism of action
As mentioned earlier, this medication contains both fluticasone and salmeterol. Both mechanisms of these drugs are different since they belong to different drug families. However, their effects combine and potentiate each other.
Fluticasone Propionate is a synthetic corticosteroid, so it has mainly anti-inflammatory activity, although it also acts by other routes. It is more effective than other corticosteroids prescribed in asthma or COPD, such as betamethasone and dexamethasone; experts say that it could be up to 18 times more effective. Studies show that it is up to 3 times more effective than budesonide. Very important data and of great relevance when it comes to clinical significance, given that it shows a much greater potential.
Inflammation is a basic component in triggering asthma and COPD. Corticosteroids act very well on this pathophysiological basis, as their effect occurs on almost all cell types, by decreasing inflammatory reactions and, especially, white blood cells and mediators such as leukotrienes and histamine.
In the case of COPD, corticosteroids are not widely used to treat the condition as such. However, Advair Diskus is an exception to the rule and can be used in this pathology.
This is a Beta-Agonist, one of the classes of drugs most used in the respiratory and cardiovascular part. Salmeterol belongs to the LABA (Long Action Beta-Agonist) family, one of the fundamental treatment lines in Asthma. According to many available studies, salmeterol appears to be up to 50 times more effective for beta-receptors than other drugs in its family.
Although Beta-Agonists’ effect on B2 receptors is often related to the adrenergic system, such as vascular contraction that gives way to hypertension, their action is quite different in the bronchial tree.
Bronchi have smooth muscle which reacts to beta-agonists from bronchodilation. It is possible that, due to the presence of B1 receptors in the heart, its rate may be accelerated with the use of medication; this is a frequent sign, important in people with cardiac pathologies.
The direct bronchodilator effect of Beta 2 drugs, such as salmeterol, is due to the action of cyclic AMP, a secondary receptor found inside cells. This allows the bronchi to widen and the amount of air passing through the airways is much greater.
In the case of asthma, what happens is that the airways become very narrow, making it difficult for air to reach the alveoli (where breathing actually occurs), which gives rise to the "whistles" characteristic of asthma. On the other hand, when the bronchi are relaxed, the flow is much greater and it is possible to avoid respiratory crises.
In addition, other important studies have shown that Salmeterol has a secondary effect on mast cells, the cells that mediate allergic reactions and part of inflammatory reactions. In this way, the release of histamine, leukotrienes and prostaglandin D2 is inhibited, attenuating allergic responses and bronchial hyper-reactivity.
As mentioned above, it can become a key piece in the treatment of asthma. It is used in patients over 4 years old.
The most important and contributing part of asthma treatment is Salmeterol, the LABA agent. This active ingredient allows the expansion of the airways, and with it, a better airflow.
Many studies confirm that, as part of Advair Diskus, salmeterol greatly decreases the risk of asthma-related death. However, some studies claim that it may increase the risk of hospitalization, but not in all patients, just in children. It is important for physicians to be especially careful about prescribing this drug in those under 18, especially when they can be better controlled with other drugs.
In addition, we must remember that this is not a rescue drug but serves as a treatment in the prevention of asthmatic crises. For this reason, it is important for you to know that you should NOT use it during active crises, but after they have been resolved. If you have a crisis, it is best to go to the doctor or use the prescribed medications to treat the crisis.
After you have controlled your asthma, you may begin treatment with Advair Diskus; however, you must remain attached to it in order to achieve effective control of this pathology.
Chronic obstructive pulmonary disease (COPD) can be difficult to treat in many cases, especially emphysema. In the case of this pathology, it should ideally be used as maintenance rather than rescue treatment, in the same way as with asthma.
Advair Diskus effectively reduces exacerbations in these patients, especially those who frequently suffer from these crises.
The initial treatment is based on two daily doses so that it is adequately impregnated and can have the desired effect. Whether it is chronic bronchitis or emphysema, Advair Diskus has proven efficacy and many studies support it.
Dosage and Presentation
Doctors usually prescribe an inhalation twice a day. However, they may have different indications depending on the condition. The first number corresponds to Fluticasone, and the second to Salmeterol.
- Advair Diskus 100/50
- Advair Diskus 250/50
- Advair Diskus 500/50
Remember, it can only be used as a maintenance treatment and not as a rescue treatment.
For adult patients (includes those over 12 years old):
One inhalation a day, twice a day. Take special care to cover the hours properly, try not to skip the intervals or separate them.
However, doses may be adjusted according to patient need and clinical severity. The maximum Advair Diskus that you can dispose of daily is 500/50 twice a day.
Since this is a corticosteroid therapy, you will most likely not see immediate results but will have to wait at least a week to see the effects. Let us remember that being maintenance, we do not look for a quick resolution of your problems but we do not want them to reappear again. Even so, we all react differently to medications, so it is likely that this time will vary in you.
If there are no tangible results in two weeks, it is best to change therapy. The components of Advair Diskus are probably not ideal for you.
In patients between 4 and 12 years old, they should require the same style of treatment, but with lower dose presentations. Use Advair Diskus 100/50 twice a day.
The basis of the treatment in terms of dosage is the same, the problem is that it must be used effectively, analyzing the patient well, in order to determine whether he is under crisis or not. There are some cases where patients have slight decreases in oxygen saturation, so it does not manifest as a major crisis, but remains one.
In this case, it is best not to use this medication but to switch to some short-acting ones, such as salbutamol. After the patient comes out of the crisisinsid if possible use Advair Diskus.
In the same way, the effects will be better appreciated in the first week, and if they do not exist in two weeks, you will have to change the medicine to a more efficient one.
How to use Advair Diskus
We will go step by step explaining the proper use of this medicine. Most people do not know how to use inhalers properly.
1) Expose the mouthpiece
You must hold the inhaler with your dominant hand in a horizontal position. You must place your thumb over the special recess and move the cover outwards. When you do this, the inside of the inhaler should rotate and make a sound when it is fixed.
Near that cutout where your thumb should be placed, is a very important number. This will tell you how many doses you have left, so be aware of it. When you have a few left, it will turn red "0-5".
2) The Lever
When you expose the nozzle, there is a small lever right in front of it. Hold the inhaler horizontally and slide the lever until you hear a small click. What the inhaler does is prepare a new dose inside the inhaler, so that you can release it when you put it in your mouth.
Inside it, there are many small packages filled with the medication. When you push the lever, it breaks one of the packs and the medicine is exposed.
Now, before you finally put the inhaler in your mouth, you should exhale as much as you can and try to empty your lungs as much as you can so that the medicine reaches the whole lung better when you inhale.
Try to keep the inhaler far away at this time, so that you do not contaminate it and do not miss the dose by blowing.
Place the inhaler in your mouth and take a deep breath. Some recommend that you take an inhalation as deep as you can in a second, so that the medicine does not lodge in the airways, but quickly reaches the depths of your lungs. Remember, you must inhale through your mouth and always keep the inhaler horizontal.
Hold your breath for 15 seconds to make sure the medicine is absorbed properly. If you can hold your breath longer, so much the better. After that, you can breathe quietly again.
6) Rinse out
Since breathing is through the mouth, when you finish inhaling you must eliminate the medicine that is around your mouth. To do this, make several gargles with water, without swallowing it, and spit it out.
Since the drug is an immunosuppressant (it reduces inflammatory reactions and defenses), you may become prone to oral infections if Advair Diskus remains in your mouth. So be sure to get rid of it.
Like most medicines, it is possible to witness unwanted effects that may become important. As mentioned above, Advair Diskus may even increase the risk of asthma-related death in some cases. Some of the possible side effects are:
Fungal infections are mostly opportunistic. This means that they expect the guest to have low defenses and not be able to attack while they are colonizing. When the corticosteroids that are part of the Advair Diskus remain in the mouth for a long time, they can cause this type of immunosuppression, which will later facilitate the colonization of secondary fungal infections, such as Candida albicans.
If so, you will notice some abnormal lesions in your mouth. See your doctor promptly and stop using the medication. The lesions should be treated appropriately and the type of drug you are using should be changed.
It occurs mainly in COPD patients and follows the same pathophysiological mechanism mentioned above, but mainly of bacterial origin.
Similarly, in COPD (especially in chronic bronchitis) large accumulations of mucus can be a great settlement for the colonization of certain bacteria. Therefore, they are prone to suffer from pneumonia constantly. Corticosteroids only facilitate this state by lowering the defenses, so the frequency of pneumonia can be quite high.
Other less frequent side effects are:
- Generalized immunosuppression
- Adrenal suppression
- Decreased bone density
- Muscle injuries
- Sleep disorder
Bronchial asthma is a dangerous disease
Bronchial asthma (BA) is a chronic inflammatory disease of the respiratory tract, the obligatory mechanism of which is bronchial hyperreactivity and reversible bronchial obstruction caused by acute bronchospasm, edema of the walls of the bronchi, the formation of mucus membranes with subsequent reconstruction of the walls of the bronchi.
Predisposing factor for asthma is allergy.
In recent decades, the problem of asthma has become one of the most pressing in medicine for a number of reasons:
- an increase in the incidence and weighting of the disease, especially in children and adolescents;
- early disability;
- increased mortality.
Along with the medical factors of this alarming statistics (heredity, the presence of chronic bacterial and viral infections, parasitic invasions, impaired immune system functions, smoking, unhealthy diet, uncontrolled use of many drugs) and objective (bad ecology), late diagnosis and inadequate play a significant role therapy. The reasons for the latter are numerous, both from the side of medicine and from the side of patients.
Medical and social aspects:
- errors in diagnosis,
- lack of specialized assistance and diagnostic capabilities,
- violations of the provision of free drugs,
- underestimation of the severity of the condition
- fear of prescribing corticosteroid drugs
- lack of control of conducting and educational programs for patients.
- late appeal
- self-medication, especially the uncontrolled use of bronchodilator drugs, which is one of the main causes of death in asthma,
- underestimation of the severity of the condition
- fear of corticosteroid treatment,
- failure to attend check-ups to the doctor, unwillingness to participate in educational programs.
As you can see, the fault of doctors and patients coincides on many points.
BA can occur at any age, most often after a respiratory infection. In most cases, the development of asthma attacks for several years preceded by allergic rhinitis, conjunctivitis, unproductive cough. The frequency of attacks depends on the severity of the disease, but I want to especially emphasize that asthma of any severity requires examination and treatment. Like any chronic disease, asthma requires adequate treatment in the period of exacerbation and prophylaxis in the period of remission.
Medical standards for the diagnosis and treatment of patients with bronchial asthma
- Mandatory laboratory tests:
- Clinical blood test (1 time in 10 days);
- Biochemical blood test (bilirubin, ALT, ACT, urea, glucose);
- RW, HIV;
- Common sputum analysis;
- Bacteriological examination of sputum for flora and sensitivity to antibiotics;
- Urinalysis common.
- Additional laboratory tests:
- Determination of protein fractions;
- Determination of the level of IgA, IgM, IgG in serum;
- Blood test: cortisol, ACTH (for patients receiving systemic corticosteroids);
- Analysis of urine for the content of 17- (for patients receiving GKS of systemic action).
- An allergic examination is mandatory:
- Skin tests with allergens;
- Determination of the level of total IgE;
- Determination of the level of specific IgE.
- Allergological examination additional:
- Provocative tests with allergens (conjunctival, nasal, inhalation);
- Determination of the level of specific IgG;
- The test of inhibition of natural emigration of leukocytes.
- Mandatory instrumental studies:
- Radiography of the chest;
- X-ray of the paranasal sinuses;
- ECG 1 time (in case of pathology - repeatedly);
- study of respiratory function 2 times.
- Additional instrumental studies:
- Diagnostic bronchoscopy;
- Bronchomotor tests (assessment of respiratory function after exposure to bronchodilators, bronchoconstrictors, physical activity).
- Mandatory expert advice:
- Additional expert advice - on the testimony.
Recent studies have confirmed the indisputable link between severe asthma and the presence of chronic, often recurring, infectious and inflammatory diseases, sluggish, difficult to treat, caused by intracellular microorganisms (chlamydia, mycoplasma), viruses (cytomegalovirus, Epstein-Barr virus, herpesvirus), worms. In connection with the need for rehabilitation of foci of chronic infection and correction of the immune status (in the presence of clinical signs of impaired immunity), it is necessary:
- study of the immune status with the control after the treatment;
- virological examination;
- diagnostics of parasitic invasions (analysis of feces on helminth eggs, detection of antibodies to helminths).
Basic principles of treatment of bronchial asthma
1. Termination or reduction of contact with allergens: air cleaners, air conditioners, humidifiers, fight with dust mites, cockroaches, special covers for bedding, abandonment of carpets, keeping pets, hypoallergenic diet and other activities.
2. Drug therapy: determined by the degree of severity of the disease, appointed taking into account the complications of the underlying disease and the presence of comorbidities.
3. Allergen-specific immunotherapy - ASIT - is the main treatment for bronchial asthma. Indications for ASIT in patients with atopic BA:
- clear confirmation of the role of the allergen in the development of the disease (house dust mites, household allergens, plant pollen, fungi);
- confirmation of the IgE-dependent sensitization mechanism;
- the inability to stop the contact of the patient with allergens;
- age from 5 to 50 years. Contraindications for ASIT:
- exacerbation of asthma;
- severe BA;
- oncological, autoimmune, mental diseases, blood diseases;
- acute infectious diseases;
- chronic infections in the acute stage.
The purpose of this method is to reduce the sensitivity to a causally significant allergen, affecting the immune mechanism like vaccination. The safety and effectiveness of ASIT has been proven by the many years of application of the method of medicine in all developed countries. Duration of treatment is 3-5 years. Treatment courses can be:
- short preseason;
- full preseason;
The selection of allergens and the course of treatment is determined only by the allergist and doctor in a medical institution. Allergens (or a mixture of 2-3 allergens) are administered as subcutaneous injections or sublingually in drops (for children) in micro doses, according to standard or individual schemes, with increasing doses and concentrations of allergens. The expected effects of immunotherapy:
- ASIT prevents BA from becoming more severe;
- reduced need for the volume of drug therapy;
- leads to remission of the disease (up to several years);
- prevents the expansion of the spectrum of allergens.
The likelihood of a positive effect increases if:
- treatment is started as early as possible, optimally - at the stage of allergic rhinitis or when the first “signals” of BA appear - lung, single attacks of asphyxiation;
- all foci of chronic infections were sanitized;
- antihelminthic therapy, correction of the immune status, if necessary.
The role of the patient in the treatment of asthma
As with other chronic diseases that require almost daily monitoring (for example, diabetes, hypertension), success in therapy, without exaggeration, depends half on the patient. Suppose you have found a doctor you trust, you are correctly diagnosed, treatment is prescribed. But this is only the beginning. And then what? Then - a course to reduce symptoms to a minimum, to improve the quality of life, to achieve long-term remission. For this, apart from desire, awareness of the disease and treatment, punctuality and patience are important. 5 tips for patients with asthma:
- Learn about your illness from a doctor, from popular science literature, and not from neighbors and colleagues. With similar symptoms, you may have completely different diseases. With the same diagnosis, you may be contraindicated drugs prescribed to another asthma.
- Visit the Asthma School. Spend at home all the recommended activities. It often helps to reduce the frequency and intensity of attacks. If it is impossible to get rid of household allergens at all, then partially - it is quite capable.
- Do not change the prescribed treatment regimens, dosages of drugs on their own, especially - do not cancel the basic therapy!
- Visit the doctor, both for the scheduled follow-up examination and for exacerbation of the disease.
- Monitor your condition and the effectiveness of drug therapy using daily peak flowmetry.
No matter how trite, but I want to remind you of the important: food, feasible sports, working conditions, rest, sanatorium-resort treatment. If there is a disease - it is time to think about it.
Where and who is treated, everyone decides for himself. It is a matter of personal responsibility for one’s health to choose a doctor or healer, to self-medicate. By contacting us, you have the opportunity to complete a survey in accordance with the necessary standards within 1 day, to make or clarify the diagnosis as soon as possible, to receive adequate treatment and detailed answers to all your questions.
Today we have all the possibilities for this. And yet the main thing: success in the treatment of bronchial asthma - in the long-term, confidential and effective union of the doctor and the patient.
COPD: Answers to the most important questions
COPD is a widely known diagnosis in people over 45 years of age. It affects the lives of 20% of the adult population of our planet. COPD is ranked 4th on the list of leading causes of death among middle-aged and elderly people. One of the most dangerous features of this disease is its inconspicuous onset and gradual, but steady development. The first ten years of the disease, as a rule, fall out of sight and the sick and doctors. The obvious symptoms of the development of a serious and dangerous disease for many years are taken as the natural consequences of colds, bad habits and age-related changes. Staying in such delusions, a sick person for years avoids the question of diagnosing and treating his illness. All this leads to almost irreversible progress of the disease. A person gradually loses working capacity, and then does the opportunity to fully live. There is a disability.
Diagnosis of copd - what is it?
COPD stands for Chronic Obstructive Pulmonary Disease. The disease is characterized by chronic inflammation in the lungs with a constantly progressive decrease in airway patency. A provocateur of such inflammation is regular inhalation of tobacco smoke, as well as household and industrial chemicals from the surrounding air.
Regularly inhaled irritants cause chronic inflammation in the airways and lung tissue. As a result of this inflammation, two pathological processes develop simultaneously: constant edema and narrowing of the airways (chronic bronchitis) and deformation of the lung tissue with loss of its function (emphysema). The combination of simultaneously occurring and developing these processes and their consequences is the chronic obstructive pulmonary disease.
In turn, the leading provocateurs of COPD development are smoking, working in hazardous production with constant inhalation of irritants and serious air pollution from the products of fuel combustion (life in the megalopolis).
How to recognize copd? The beginning and leading symptoms of the disease
Chronic obstructive pulmonary disease develops gradually, starting with the smallest symptoms. For many years, a sick person considers himself "healthy." The main difference of the disease is its steady, poorly reversible progress. Therefore, often, the patient turns to the doctor already having reached the disabling stage of the disease. However, there are three main reasons to suspect COPD at almost any of its stages:
- the presence of a regularly inhaled pathogen (smoking, manufacturing, etc.)
- cough / sputum
- appearance of noticeable shortness of breath after exercise
As a rule, the disease begins with the appearance of cough. Most often it is a cough in the morning, with sputum discharge. The patient has the so-called "frequent colds." Most of all, such a cough worries in the cold season - the autumn-winter period. Most often in the early years of the onset of COPD, patients do not associate a cough with an already developing disease. Cough is perceived as a natural smoking companion that does not pose a health hazard. While this cough may be the first alarm signal in the development of a difficult and almost irreversible process.
There is noticeable shortness of breath at the beginning of climbing stairs and brisk walking. Patients often take this state as a natural result of the loss of their former physical form - exercise. However, dyspnea in COPD progresses steadily. Over time, less physical exertion causes a lack of air, a desire to catch my breath and stop. Up to the onset of dyspnea even at rest.
Aggravation of COPD
The most dangerous, periodic complication of the disease. In the overwhelming percentage of cases, the exacerbation of symptoms of COPD occurs against the background of bacterial and viral infections of the upper respiratory tract. Especially often this occurs in the autumn-winter period of the year, during the seasonal jump in the viral incidence of the population.
Exacerbation is manifested by a significant deterioration in the patient’s condition, which lasts more than a few days. There is a noticeable increase in cough, changes in the amount of sputum released with cough. Increase in shortness of breath. At the same time, the respiratory function of the lungs is significantly reduced. The worsening of symptoms during COPD exacerbations is a potentially life-threatening condition. An exacerbation can lead to the development of severe respiratory failure and the need for hospitalization.
How to distine copd from asthma and other diseases?
There are several main signs that allow to distinguish between COPD and bronchial asthma even before the examination. So with COPD: consistency of symptoms (cough and shortness of breath), the presence of a regularly inhaled pathogen (smoking, manufacturing, etc.), the age of the patient is over 35 years old.
Thus, clinically, COPD differs from asthma, above all, by the persistence of symptoms over a long time. Asthma is characterized by a bright, undulating course - attacks of lack of air are replaced by periods of remission.
In COPD, it is almost always possible to find a constant provoking respiratory factor: tobacco smoke, participation in hazardous production.
Finally, COPD is a disease of the adult population - middle-aged and elderly people. At the same time, the older the age, the more likely the diagnosis of COPD is in the presence of characteristic symptoms.
Of course, there are a number of instrumental and laboratory studies that allow you to determine the diagnosis of COPD. The most significant among them are: respiratory tests, blood and sputum examination, x-ray of the lungs and ECG.
Why is copd dangerous? What does this disease conduct?
The most dangerous feature of COPD is the subtle and gradual progression of the disease. Already a sick person, considering himself “practically healthy” for 10–15 years does not pay the necessary attention to his condition. All symptoms of the disease are attributed to weather, fatigue, age. During all this time, COPD continues to progress steadily. Progress until it becomes impossible to notice the disease.
Disability A patient with COPD gradually loses the ability to endure physical exertion. Climbing the stairs, brisk walking becomes a problem. After such loads, the person begins to choke - there is a strong shortness of breath. But the disease continues to develop. So, gradually going to the store, minor physical exertion - all this now causes respiratory arrest, severe shortness of breath. The final of the disease started - complete loss of exercise tolerance, disability and physical disability. Severe shortness of breath even at rest. It does not allow the patient to leave the house and fully serve themselves.
Infectious exacerbations of COPD. Virtually any upper respiratory tract infection (eg, flu), especially during the cold season, can lead to severe exacerbation of the symptoms of the disease, up to hospitalization in intensive care with severe respiratory failure and the need for artificial respiration.
Irreversible loss of heart function - "pulmonary heart." Chronic stagnation in the pulmonary circulation, excessive pressure in the pulmonary artery, increased load on the heart chambers - almost irreversibly alter the shape and functionality of the heart.
Cardiovascular Diseases acquire the most aggressive and life-threatening course against the background of COPD. The patient's risk of developing coronary heart disease, hypertension and myocardial infarction increases significantly. At the same time, concomitant cardiovascular diseases themselves acquire a severe, progressive and poorly treatable course.
Atherosclerosis of the vessels of the lower extremities - most often occurs with COPD. It is a change in the vessel wall with subsequent deposition of cholesterol plaques, impaired patency and the risk of pulmonary embolism (PATE).
Osteoporosis (increased bone fragility) occurs in response to a chronic inflammatory process in the lungs.
Progressive muscle weakness - the gradual atrophy of skeletal muscles almost always accompanies the progress of COPD.
Based on the above consequences of the progress of COPD, its features, as well as the conditions accompanying it, the most dangerous for the patient’s life complications arise, most often leading to death:
- Acute respiratory failure - the result of exacerbation of the disease. Extremely low oxygen saturation, life-threatening condition requiring immediate hospitalization.
- Lung cancer is the result of the patient’s lack of alertness regarding his illness. The result of underestimating the risk of constant exposure to risk factors and the lack of measures taken for the timely diagnosis, treatment and modification of lifestyle.
- Myocardial infarction is a frequent complication of coronary heart disease associated with COPD. The presence of COPD doubles the risk of heart attack.
- Treatment of COPD: the main options and their prospects.
First of all, it is necessary to understand: neither medicine nor surgery can cure the disease. They temporarily restrain her symptoms. Drug therapy for COPD is a lifelong inhalation of agents that temporarily expand the bronchi. In the case of diagnosis of the disease in the middle and severe stages, glucocorticosteroid hormones are added to the above drugs, designed to strongly contain chronic inflammation in the airways and temporarily reduce their swelling. All these drugs, and in particular drugs based on glucocorticosteroid hormones, have a number of significant side effects that significantly limit the possibilities of their use in different categories of patients.
How to stop copd without drugs?
The first thing you need to understand for every patient with COPD: quitting smoking is absolutely necessary. The treatment option for the disease without eliminating the inhaled irritant is impossible. If the cause of the disease is harmful production, inhalation of chemicals, dust - in order to save health and life, it is necessary to change working conditions.
Back in 1952, Soviet scientist Konstantin Pavlovich Buteyko developed a method that, without the use of drugs, would significantly alleviate the condition of patients with officially recognized "incurable" disease - COPD.
Dr. Buteyko’s research has shown that the patient’s breathing depth makes a huge contribution to the development of bronchial obstruction, the formation of allergic and inflammatory responses.
Excessive depth of breathing is deadly for the body, it destroys the metabolism and the normal course of a number of vital processes.
Buteyko proved that the patient's body automatically protects itself from excessive depth of breathing - natural defensive reactions occur, aimed at preventing the outflow of carbon dioxide from the lungs. So there is swelling of the mucous membrane of the respiratory tract, smooth muscles of the bronchi are compressed - all this is natural protection against deep breathing.
It is these protective reactions that play a huge role in the course and development of such pulmonary diseases as asthma, bronchitis and COPD. And every patient is able to remove these protective reactions! Without the use of any drugs.
Buteyko breathing exercises are a universal way to normalize breathing, designed to help patients with the most well-known pathology. Assistance that does not require drugs and surgery. The method is based on the revolutionary discovery of deep-breathing diseases, accomplished by Dr. Buteyko as far back as 1952. For more than thirty years, Konstantin Pavlovich Buteyko devoted to the creation and detailed practical development of this method. Over the years, the method has helped save the health and lives of thousands of patients. The result was the official recognition of the Buteyko method by the Ministry of Health of the USSR on April 30, 1985 and its inclusion in the standard of clinical therapy of bronchopulmonary diseases.